green fluorescent protein fusion (OriGene)
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OriGene
green fluorescent protein fusion
Green Fluorescent Protein Fusion, supplied by OriGene, used in various techniques. Bioz Stars score: 94/100, based on 6 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/green+fluorescent+protein+gfp/pLenti-EF1a-C-mGFP+Lentiviral+Gene+Expression+Vector/pm42271190-47-26-29
Average 94 stars, based on 6 article reviews
Green Fluorescent Protein Fusion, supplied by OriGene, used in various techniques. Bioz Stars score: 94/100, based on 6 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/green+fluorescent+protein+gfp/pLenti-EF1a-C-mGFP+Lentiviral+Gene+Expression+Vector/pm42271190-47-26-29
Average 94 stars, based on 6 article reviews
green fluorescent protein fusion - by Bioz Stars,
2026-10
94/100 stars
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Related Articles
Plasmid Preparation:Article Title: Dynamics of ADAM17-Mediated Shedding of ACE2 Applied to Pancreatic Islets of Male db/db Mice. Article Snippet: .. A plasmid for a fusion protein of human ACE2 and Transfection:Article Title: Sarcolemmal loss of active nNOS (Nos1) is an oxidative stress-dependent, early event driving disuse atrophy. Article Snippet: Skeletal muscle atrophy following unloading or immobilization represents a major invalidating event in bedridden patients.. Among mechanisms involved in atrophy development, a controversial role is played by neuronal-NOS (nNOS; NOS1) whose dysregulation at the protein level and/or subcellular distribution also characterizes other neuromuscular disorders.. This study aimed to investigate unloading-induced changes in nNOS before any evidence of myofiber atrophy, using vastus lateralis biopsies obtained from young healthy subjects after a short bed-rest and rat soleus muscles after exposure to short-unloading periods. shRNA:Article Title: Sarcolemmal loss of active nNOS (Nos1) is an oxidative stress-dependent, early event driving disuse atrophy. Article Snippet: Skeletal muscle atrophy following unloading or immobilization represents a major invalidating event in bedridden patients.. Among mechanisms involved in atrophy development, a controversial role is played by neuronal-NOS (nNOS; NOS1) whose dysregulation at the protein level and/or subcellular distribution also characterizes other neuromuscular disorders.. This study aimed to investigate unloading-induced changes in nNOS before any evidence of myofiber atrophy, using vastus lateralis biopsies obtained from young healthy subjects after a short bed-rest and rat soleus muscles after exposure to short-unloading periods. Construct:Article Title: Sarcolemmal loss of active nNOS (Nos1) is an oxidative stress-dependent, early event driving disuse atrophy. Article Snippet: Skeletal muscle atrophy following unloading or immobilization represents a major invalidating event in bedridden patients.. Among mechanisms involved in atrophy development, a controversial role is played by neuronal-NOS (nNOS; NOS1) whose dysregulation at the protein level and/or subcellular distribution also characterizes other neuromuscular disorders.. This study aimed to investigate unloading-induced changes in nNOS before any evidence of myofiber atrophy, using vastus lateralis biopsies obtained from young healthy subjects after a short bed-rest and rat soleus muscles after exposure to short-unloading periods. other:Article Title: Carboplatin alleviates astrocytic TDP-43 neurotoxicity by inhibiting NF-κB activation. Article Snippet: Amyotrophic lateral sclerosis (ALS) is a rare and progressive motor neuron disease; however, its exact pathogenic mechanisms remain unclear.. Currently, no effective treatments are available for this disease.. Therefore, in this study, we investigated the anti-inflammatory effects of the anti-cancer agent, carboplatin, on neuronal cells and its potential therapeutic effects against ALS. |